Prevent Acne Scars By Taking Proper Care Of Your Pimples

You must start the treating of acne abrasions before having to regret the loss of tissues (holes) or the surplus fibrotic scarring your system creates when it reacts with a strong agitative response, for then acne scar treatment isn't going to be straightforward or fast.

Facial acne scars are characterized either by loss of tissue (ice pick or pitted, indented boxcar scars, rolling acne scars) or by the abnormal formation of new thickened tissues. Those acne punctures are wounds caused to the cells lining the sebum canals when there is a sudden outflow of sebum to the outer layer of the skin caused by hormonal disequilibria or because of increased strain on those cells stemming from blockage of such outflow by clogged or blocked pores.

In fact acne scarring is the result when our immune system has to rely just on its own resources to fix acne punctures, for then it acts in accordance with the evolutionary lesson it learned during our evolutionary drift: All wounds or injuries, scrapes or blisters represent a perilous threat to our survival, not only due to blood loss, but also due to tissue damage or infection from the invasion of micro-organisms and/or foreign bodies like mud, splinters and bacteria.

The healing mechanism that has developed to respond to such threats has 2 basic characteristics:

First, there's a quick and robust inflammatory response, which sets on the alarms and triggers a cascade of events that start with an action call to its fire fighters to the injured site.

2nd, there is a fibrotic"walling-off" reply to isolate foreign bodies, and stop germs from ceasing the chance to penetrate at the location of a wound.

Problem is those fire fighters arrive inflamed with chemical weapons and can care less if they also liquefy or destroy diagonally opposite skin and lead straight to abscess formation. After all they have learned, during our evolutionary drift, that trespassers at a location of lesion may become a case of life or death.

Beauty Isn't a Priority For Our Immune Mechanism

To restore the integrity of the skin as fast as possible closing of skin punctures and repairing of missing skin tissues happen rapidly by an evolutionary mechanism that leads to granulation and fibrosis, and in other commoner words: acne scars.

By contrast, when granulation tissues form in wound healing, collagen bundles are laid down in parallel between the margins of the skin lacerations. The abnormal design of the collagen deposition creates tissues of weaker tensile strength and produces the characteristic scars.

Studies of the wound healing process in humans and in animals show, for certain, that the first 48 hours after a skin lesion are key in determining the scarring end result. The best outcome is when interventions are made within this window. A possible explanation is that the low number of signaling molecules in the opening cytokine cascade caused by the skin curing process can have a deep effect on the levels/proportions of inflammatory cells and growth factors inducted to the wound site. In addition, the recruited cells influence the receptor profiles on the target cells, further impacting on the healing reply and successive scar deposition.

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